PD Dr. Marko Bertog

Lehrstuhl für Physiologie (Vegetative Physiologie)

Prostaglandin E2 stimulates the epithelial sodium channel (ENaC) in cultured mouse cortical collecting duct cells in an autocrine manner (2020) Mansley M, Niklas C, Nacken R, Mandery K, Gläser H, Fromm M, Korbmacher C, Bertog M Journal article Ubiquitination of renal ENaC subunits in vivo (2020) Frindt G, Bertog M, Korbmacher C, Palmer LG Journal article Aldosterone-independent ENaC activity in the early part of the aldosterone-sensitive distal nephron critically depends on the presence of mineralocorticoid receptor (2019) Nesterov V, Bertog M, Canonica J, Hummler E, Korbmacher C Conference contribution Activation of the protease-activated receptor type 2 (PAR2) has a sustained stimulatory effect on the epithelial sodium channel (ENaC) in mCCDcl1 mouse cortical collecting duct cells (2019) Schmidt M, Korbmacher C, Bertog M Conference contribution Modulatory effects of apical and basolateral proteases on renal ENaC function (2019) Bertog M, Korbmacher C Conference contribution Plasma kallikrein activates the epithelial sodium channel in vitro but is not essential for volume retention in nephrotic mice. (2018) Härteis S, Schork A, Doerffel T, Bohnert BN, Nacken R, Woern M, Xiao M, et al. Journal article Inhibitors of the proteasome stimulate the epithelial sodium channel (ENaC) through SGK1 and mimic the effect of aldosterone (2018) Mansley M, Korbmacher C, Bertog M Journal article Aprotinin prevents proteolytic epithelial sodium channel (ENaC) activation and volume retention in nephrotic syndrome (2018) Bohnert BN, Menacher M, Janessa A, Woern M, Schork A, Daiminger S, Kalbacher H, et al. Journal article Lithocholic acid has a sustained stimulatory effect on the epithelial sodium channel (ENaC) in mouse late distal colon (2017) Bertog M, Reeh L, Bunnett NW, Korbmacher C Conference contribution Activation of the Epithelial Sodium Channel (ENaC) by Aldosterone in mCCD(cl1) Mouse Renal Cortical Collecting Duct Cells is Dependent on SGK1 and Can be Prevented by Inhibiting Endogenous Proteases (2017) Korbmacher C, Mansley M, Bertog M Conference contribution