apl. Prof. Dr. Elisabeth Sock

Medizinische Fakultät Lehrstuhl für Biochemie und Pathobiochemie

Replacement of mouse Sox10 by the Drosophila ortholog Sox100B provides evidence for co-option of SoxE proteins into vertebrate-specific gene-regulatory networks through altered expression. (2010) Sock E, Wegner M Journal article, Original article Replacement of related POU transcription factors leads to severe defects in mouse forebrain development. (2009) Sock E, Reiprich S, Müller J, Stolt C, Wegner M Journal article, Original article Sox12 deletion in the mouse reveals non-reciprocal redundancy with the related Sox4 and Sox11 transcription factors. (2008) Wegner M, Sock E Journal article, Original article Anterior segment dysgenesis in the eyes of mice deficient for the high-mobility-group transcription factor Sox11. (2008) Sock E, Wegner M Journal article, Original article Prolonged glial expression of Sox4 in the central nervous system leads to architectural cerebellar defects and ataxia. (2007) Sock E, Wegner M Journal article, Original article SoxD proteins influence multiple stages of oligodendrocyte development and modulate SoxE protein function. (2006) Sock E, Wegner M, Stolt C Journal article, Original article Impact of transcription factor Sox8 on oligodendrocyte specification in the mouse embryonic spinal cord. (2005) Sock E, Wegner M, Stolt C Journal article, Original article The high-mobility-group transcription factor Sox8 is a negative regulator of osteoblast differentiation. (2005) Sock E, Wegner M Journal article, Original article The POU protein Oct-6 is a nucleocytoplasmic shuttling protein. (2005) Sock E, Wegner M Journal article, Original article Gene targeting reveals a widespread role for the high-mobility-group transcription factor Sox11 in tissue remodeling. (2004) Sock E, Wegner M Journal article, Original article